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Electrolyte marketing has turned a fairly narrow physiological question into a shelf full of products. The underlying facts are simpler: you lose sodium in sweat, the amount varies enormously between riders, and the link between that loss and the cramp you got at kilometre 140 is far weaker than the industry suggests.
This piece separates the three things that get conflated — what electrolytes do, why cyclists cramp, and what is actually worth drinking.
Exercise-associated hyponatraemia is a genuine medical emergency, and sodium intake interacts with blood pressure medication and several kidney and heart conditions. Nothing here is a dosing recommendation. If you have high blood pressure, kidney disease, or have ever collapsed or become confused during an event, talk to a doctor before changing what you drink.
What electrolytes actually do
Electrolytes are minerals that carry an electrical charge in body fluid — principally sodium, plus chloride, potassium, calcium and magnesium. They govern how fluid is distributed between your blood and your cells, and they're central to how nerve signals travel and how muscle fibres contract.
Sodium is the one worth thinking about on a bike, for one reason: it is by far the most abundant electrolyte in sweat. Potassium and magnesium losses in a normal ride are small relative to what you carry and what you eat. If a drink's headline feature is its magnesium content, that is a marketing decision, not a physiological one.
How much sodium you actually lose
Here is the number that makes generic advice useless. Whole-body sweat sodium concentration in athletes runs roughly 10 to 70 mmol per litre, with local measurements spanning about 10 to 90 — a sevenfold to ninefold spread between individuals. Two riders side by side in the same conditions can lose wildly different amounts of salt.
| Your sweat | 0.5 L/h sweat | 1.0 L/h sweat | 1.5 L/h sweat |
|---|---|---|---|
| Low sodium sweat 20 mmol/L | 230 | 460 | 690 |
| Middling 40 mmol/L | 460 | 920 | 1380 |
| Salty sweater 65 mmol/L | 750 | 1500 | 2240 |
A rough guide only, calculated from published concentration ranges (1 mmol sodium ≈ 23 mg). Your own figures depend on heat, humidity, acclimatisation, clothing and intensity — and change across a season. Treat the spread, not any single cell, as the message.
Notice the range: somewhere between roughly 200 mg and 2,200 mg of sodium an hour depending on who you are and how hard the day is. That's why a fixed recommendation — "one tablet per bottle" — is meaningless. It is either too much or too little for almost everybody.
The cramp question
Exercise-associated muscle cramp is the reason most riders buy electrolytes in the first place. It is also the claim with the weakest evidence behind it. Two competing explanations have been argued over for decades, and they make different predictions.
Dehydration / electrolyte depletion
The older, more intuitive idea
Sweating heavily depletes body water and sodium, which destabilises the muscle and triggers cramp.
Points in favour
- Cramp-prone athletes often do have saltier sweat
- Salt supplementation helps some individuals in field reports
- Fits the everyday experience of cramping on hot days
Problems with it
- Cramping athletes are frequently no more dehydrated than non-cramping ones in the same race
- Blood sodium is often normal — sometimes lower in the riders who don't cramp
- Cramp is usually confined to the working muscles, not body-wide as a systemic deficit would predict
Altered neuromuscular control
The currently better-supported model
Muscle fatigue disturbs the balance between excitatory muscle-spindle activity and inhibitory Golgi tendon organ feedback, leaving the motor neurone pool hyperexcitable.
Points in favour
- Cramp clusters in muscles that are fatigued and working at short lengths
- Strongly predicted by going faster than you trained for, or by a recent history of cramping
- Passive stretching — which loads the Golgi tendon organ — reliably stops an active cramp
Problems with it
- Doesn't explain everything: some riders clearly respond to sodium
- Mostly built on observational and electromyographic evidence rather than large trials
- Hard to translate into a single practical prevention protocol
The current weight of evidence leans towards the neuromuscular explanation. Reviews of the field conclude that dehydration and electrolyte depletion, on their own, do not explain most cases of cramp — and the strongest predictors are a prior history of cramping and riding harder or longer than you're trained for. That is an uncomfortable message, because it means the fix is mostly training and pacing, which nobody can sell you in a sachet.
Some honesty is required here, though: this is not settled. A subset of riders with very high sweat sodium losses do seem to respond to sodium, and the literature is mostly observational. If salt tablets have reliably worked for you over years, the evidence isn't strong enough to tell you you're wrong. It is strong enough to say they won't work for most people.
The risk that goes the other way
Over-drinking is the failure mode that actually hospitalises people. Exercise-associated hyponatraemia — blood sodium below 135 mmol/L — is caused predominantly by drinking more fluid than you lose, which dilutes the sodium in your blood. Mild cases feel like bloating, nausea and a headache; severe cases cause confusion, seizures and, rarely, death.
The international consensus on this is blunt: the primary prevention is not drinking to a schedule. Riders who force fluid down because a plan says 750 ml an hour, regardless of heat or effort, are taking the one risk in this article that can genuinely end a day in hospital. Drinking to thirst is, for the overwhelming majority of amateur riders, both safe and sufficient.
Headache with nausea, puffy fingers, feeling worse the more you drink, or any confusion or disorientation in a rider who has been taking on a lot of fluid. Stop, stop drinking, eat something salty, and get help. This is a medical emergency, not something to ride through.
So what should you actually drink?
Under ~60–75 minutes, temperate
Water is fine.
There is no electrolyte emergency in an hour's riding. Drink to thirst.
1–3 hours, or hot
Carbohydrate drink with sodium.
Roughly 300–600 mg sodium per litre covers most riders. The carbohydrate matters at least as much as the sodium.
Over 3 hours, hot, or a known salty sweater
Higher sodium, and eat.
Up to around 1,000 mg per litre if you lose a lot of salt. Real food contributes more sodium than people assume.
Any long event where you're drinking a lot
Do not out-drink your losses.
More fluid than you sweat is the one genuinely dangerous mistake in this article. Thirst is a better guide than a schedule.
If you want your own number rather than a range, the crude home method still works: weigh yourself naked before and after a hard hour, add back the weight of anything you drank, and the difference is roughly your sweat rate. It won't tell you your sweat sodium — that needs a proper test — but it tells you whether you're a 0.5 L/h rider or a 1.8 L/h rider, which is most of what matters. Our cycling hydration strategy guide has the full protocol.
The unglamorous conclusion
- Sodium is the electrolyte that matters on a bike. The rest are largely along for the ride.
- Your losses are individual to a degree that makes generic dosing advice worthless.
- Electrolytes are unlikely to cure your cramp. Fatigue and pacing beyond your preparation are the better-supported culprits.
- Carbohydrate does more for long-ride performance than any electrolyte formulation — see fuelling long rides.
- Don't over-drink. It is the only mistake here with a serious downside.
If cramp keeps ending your long rides at the same point, the most productive thing you can change is usually the preparation rather than the bottle: more time at the duration and intensity the event actually demands. That's a training problem, and building towards it gradually — which is what Moveee's adaptive plans do as your real rides come in — will do more than any sachet.
Sources 7
Where this article summarises a study, the study itself is linked — not a write-up of it.
- 1 Baker LB Sweating Rate and Sweat Sodium Concentration in Athletes: A Review of Methodology and Intra/Interindividual Variability · Sports Medicine · 2017
- 2 Baker LB, Barnes KA, Anderson ML, et al Normative data for sweating rate, sweat sodium concentration, and sweat sodium loss in athletes: an update and analysis by sport · Journal of Sports Sciences · 2019
- 3 Schwellnus MP Cause of exercise associated muscle cramps (EAMC) — altered neuromuscular control, dehydration or electrolyte depletion? · British Journal of Sports Medicine · 2009
- 4 Miller KC, McDermott BP, Yeargin SW, et al An Evidence-Based Review of the Pathophysiology, Treatment, and Prevention of Exercise-Associated Muscle Cramps · Journal of Athletic Training · 2021
- 5 Hew-Butler T, Rosner MH, Fowkes-Godek S, et al Statement of the Third International Exercise-Associated Hyponatremia Consensus Development Conference, Carlsbad, California, 2015 · Clinical Journal of Sport Medicine · 2015
- 6 Sawka MN, Burke LM, Eichner ER, et al American College of Sports Medicine position stand: Exercise and Fluid Replacement · Medicine & Science in Sports & Exercise · 2007
- 7 Thomas DT, Erdman KA, Burke LM Position of the Academy of Nutrition and Dietetics, Dietitians of Canada, and the American College of Sports Medicine: Nutrition and Athletic Performance · Medicine & Science in Sports & Exercise · 2016
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